In response to acrylamide exposure at concentrations from 50 mg/L to 1 g/L, gst-4::gfp signals increased in a dose- and time-dependent manner in transgenic nematodes, and the signals increased faster and their peaks were higher at 25 C than at 20 C.
This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.
This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.
This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.
This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.
This RNAi was able to suppress the locomotion phenotypes found in L-AChR(gf) animals. L-AChR(gf) enhances synaptic activation of muscles by increasing L-AChR function.
In C. elegans ahr-1 (ia03) mutants, expression of a C. briggsae unc-47 promoter in SDQR/L was completely abolished, even though the cells were still present.