Mutation of
set-26 accelerated the progressive sterility of
spr-5;
set-26 double mutants were completely sterile by generations 5-8. Although
set-26 worms did not show elevated levels of H3K4me2,
spr-5;
set-26 double mutants displayed significantly higher levels of H3K4me2 at generation 4 than
spr-5 mutants.