RNAi of Y48B6A.13 enhanced the reduction of ckb-2p::GFP expression resulting from the cdc-48.2(tm659) mutation, in the presence of the ER-stress inducer tunicamycin
The lin-14(n355) mutation (a gain-of-function which removes lin-4 binding sites from the lin-14 3'UTR) partially suppresses the eif-6(RNAi)-induced accumulation of LIN-14 protein