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Page Author Comment Date
Hung, Lee, Chen, & Huang, 2010 Laura G. Vallier While up regulation of lim-7 may occur after SMF treatment, this paper refers to developmental time reduction in "lim-7 mutants" when in actuality the authors are examining a strain with the lim-7::GFP transgene tnIs6 or tnIs5 (carrying a wild-type copy of lim-7) and not a lim-7 mutant (confirmed by personal correspondence with GS Huang). The only lim-7 mutant to date is tm674 deletion, which is 100% L1 larval lethal and thus these developmental delay experiments cannot be validated beyond that stage. These comments have been communicated to the authors. 12 years ago
GE68 Surojit Sural In 'Curatorial Remarks', the WBG article link can be added: http://wbg.wormbook.org/2010/12/06/glp-1e2141-sequence-correction/ 6 years ago
slo-1 Chris Grove Comment made on behalf of Andy Golden: """ Check out Diagnosis on Netflix by the New York Times (episode 4). There is a very rare disease caused by mutations in the human KCNMA1 gene. If you study slo-1, perhaps you can help these patients. """ 5 years ago
ky289 Swagata Dey CC bases in the ky289 variant are mutated to TC which results in the truncation of the SAD-1 protein at 228th residue by a stop codon. 4 years ago
Altun ZF et al. (2015) Dev Dyn High resolution map of caenorhabditis elegans gap junction proteins. Raymond Lee The original article to which this Corrigendum refers was published in Developmental Dynamics 238: 1936–1950. In the original published version of this article the authors noticed an error on page 1947 . The authors inadvertently suggested there was a key gap junction between RIP and PVR neurons that connects the somatic and pharyngeal nervous systems in C. elegans. The corrected sentence reads: “The combination of inx‐4 and unc‐9 is notable at the sole connection between the somatic and pharyngeal nervous systems, i.e., gap junctions between RIP and I1, and also at the gap junctions between the touch neurons and PVR, which may then function in modulation of pharyngeal pumping by the touch circuitry via the synapses PVR makes onto RIP (Albertson and Thomson, 1976; Chalfie et al., 1985).” The authors regret this error. 4 years ago
lin-38 : lrp-2 Chris Grove This annotation was made in error due to a typo at curation in which the allele n761 (of lin-38) was used instead of the correct allele n671 (of lin-17). This annotation should be corrected as of the WS281 release of WormBase. 3 years ago
lin-38 : lrp-2 Chris Grove This annotation was made in error due to a typo at curation in which the allele n761 (of lin-38) was used instead of the correct allele n671 (of lin-17). This annotation should be corrected as of the WS281 release of WormBase. 3 years ago
cam-1 : lin-38 : lrp-2 Chris Grove This annotation was made in error due to a typo at curation in which the allele n761 (of lin-38) was used instead of the correct allele n671 (of lin-17). This annotation should be corrected as of the WS281 release of WormBase. 3 years ago
cam-1 : lin-38 Chris Grove This annotation was made in error due to a typo at curation in which the allele n761 (of lin-38) was used instead of the correct allele n671 (of lin-17). This annotation should be corrected as of the WS281 release of WormBase. 3 years ago
lin-38 : vang-1 Chris Grove This annotation was made in error due to a typo at curation in which the allele n761 (of lin-38) was used instead of the correct allele n671 (of lin-17). This annotation should be corrected as of the WS281 release of WormBase. 3 years ago
egl-20 : lin-38 Chris Grove This annotation was made in error due to a typo at curation in which the allele n761 (of lin-38) was used instead of the correct allele n671 (of lin-17). This annotation should be corrected as of the WS281 release of WormBase. 3 years ago
cam-1 : lin-38 Chris Grove This annotation was made in error due to a typo at curation in which the allele n761 (of lin-38) was used instead of the correct allele n671 (of lin-17). This annotation should be corrected as of the WS281 release of WormBase. 3 years ago
lin-38 : vang-1 Chris Grove This annotation was made in error due to a typo at curation in which the allele n761 (of lin-38) was used instead of the correct allele n671 (of lin-17). This annotation should be corrected as of the WS281 release of WormBase. 3 years ago
egl-20 : lin-38 Chris Grove This annotation was made in error due to a typo at curation in which the allele n761 (of lin-38) was used instead of the correct allele n671 (of lin-17). This annotation should be corrected as of the WS281 release of WormBase. 3 years ago
ser-7 Ralph Menzel I wonder if the expression of ser-7 in marginal cells is indeed correct (would be interesting for us). In the Hobson paper they do not mention marginal cells, but MCs, but they mean MC neurons and and not mc-cells as mc1, mc2 or mc3 (marginal cells). For me, also the GFP localization figure does not show marginal cells. What is true? I do not know exactly and would be happy about some advice. Ralph. 3 years ago
ser-7 Daniela Raciti Hi Ralph, Sorry for the delay in getting back to you. Indeed the annotation should have been to MC neuron and not to marginal cell. I fixed the record for Expr3759. The update will be available on WB release WS283. Many thanks for letting us know! Best, Daniela 3 years ago
cas46 Chris Grove The "neuronal cell fate specification variant" phenotype indicated for the cas46 allele with remark: "Animals exhibited extra AVM and PVM but loss of AQR and PQR. Ectopical expression of EGL-13 induced extra AQR and PQR." was the result of a faulty submission form error and should be attributed to the egl-13 allele cas11. This has been corrected in the curation database and will appear correctly in the WS283 release of WormBase, due out in late January 2022. 3 years ago
ham-1 Chris Grove The "neuronal cell fate specification variant" phenotype indicated for the cas46 allele with remark: "Animals exhibited extra AVM and PVM but loss of AQR and PQR. Ectopical expression of EGL-13 induced extra AQR and PQR." was the result of a faulty submission form error and should be attributed to the egl-13 allele cas11. This has been corrected in the curation database and will appear correctly in the WS283 release of WormBase, due out in late January 2022. 3 years ago
ca1 Chris Grove The "mRNA surveillance defective" phenotype indicated for the ca1 allele with remark: "Inefficiently degrades mRNAs lacking a stop codon" was the result of a faulty submission form error and should be attributed to the skih-2 allele cc4141. This has been corrected in the curation database and will appear correctly in the WS283 release of WormBase, due out in late January 2022. 3 years ago
ges-1 Chris Grove The "mRNA surveillance defective" phenotype indicated for the ca1 allele with remark: "Inefficiently degrades mRNAs lacking a stop codon" was the result of a faulty submission form error and should be attributed to the skih-2 allele cc4141. This has been corrected in the curation database and will appear correctly in the WS283 release of WormBase, due out in late January 2022. 3 years ago
Katoh T et al. (2013) Glycobiology Deficiency of -glucosidase I alters glycoprotein glycosylation and lifespan in Caenorhabditis elegans. James There's some confusion on the links in this abstract. The gene 'F13H10.4' is now 'mogs-1' not 'agl-1'. The links incorrectly go to gene 'R06A4.8', which is now designated 'agl-1'. Their proteins do not share sequence similarity. 172 days ago
cc546 Dave Reiner The cc546 temperature sensitive allele causes a T761I change in SMG-1. The lesion is a atg>ttg transversion in exon 35. Flanking sequences follow with the mutation site a capital A: ttggtggtcggttacaaaacgatattcaaga tcactggcagtcatgagtAtggttggatcagttttaggactcggtgatcg acatttggacaatttattg The lesion is detectable via SNP-snip with the mutation causing loss of an MslI site. Primers are for a 323 bp product. Digest with MslI to 86+237 in the wild type, uncut as 323 in the mutant. DJR701(f): CAGTCGTGAGCTTTGGATGCGTGC DJR702(r): TCGGGGATACGCAGATTCTTTCCC Pedone ... Reiner G3 (2021) 167 days ago
cc545 Dave Reiner The cc545 temperature sensitive allele causes a T761I change in SMG-1. The lesion is a aca>ata transition in exon 35. Flanking sequences follow with the mutation site a capital C: tggattattaatcagact gcaaacttttgcattgtgaataaaatgaagaCaccattaggaaaaccaat gcagacttttgcagcttttgagaatgaaatta Pedone ... Reiner G3 (2021) 167 days ago
cc546 Dave Reiner Sorry, this is an M1957L change (T761I is for cc545) 167 days ago
cc546 Dave Reiner Sorry, this is an M1957L change (T761I is for cc545) 167 days ago