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WormBase Tree Display for Variation: WBVar00090140

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Name Class

WBVar00090140NamePublic_namen1347n1348
Sequence_detailsSeqStatusPending_curation
Variation_typeAllele
OriginSpeciesCaenorhabditis elegans
LaboratoryMT
StatusLive
Linked_toWBVar00090139
WBVar00090141
AffectsGeneWBGene00001078
DescriptionPhenotypeWBPhenotype:0000104Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Remark"In the null dpy-19 allele, n1347n1348 (see below), QL showed no clear direction of polarization in 72% of animals and projected anteriorly in 8% of animals. Similarly, QR lacked clear polarization in 70% of animals and projected incorrectly towards the posterior in 8% of animals."Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Variation_effectNullPaper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
EQ_annotationsAnatomy_termWBbt:0004056PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
WBbt:0004054PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
GO_termGO:0030010PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Phenotype_not_observedWBPhenotype:0000195Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Remark"No defects in the embryonic migrations of HSN, CAN, M and ALM were seen in dpy-19(n1347n1348) animals (n=35) and no defects in the postembryonic migrations of the distal tip cells (DTCs) or male linker cell were seen in dpy-19(mu78) animals (n=50)."Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Variation_effectNullPaper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
EQ_annotationsAnatomy_termWBbt:0006865PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
GO_termGO:0016477PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
WBPhenotype:0000232Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Remark"No defects in the embryonic migrations of HSN, CAN, M and ALM were seen in dpy-19(n1347n1348) animals (n=35) and no defects in the postembryonic migrations of the distal tip cells (DTCs) or male linker cell were seen in dpy-19(mu78) animals (n=50)."Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Variation_effectNullPaper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
EQ_annotationsAnatomy_termWBbt:0006827PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
GO_termGO:0016477PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
WBPhenotype:0000470Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Remark"No defects in the embryonic migrations of HSN, CAN, M and ALM were seen in dpy-19(n1347n1348) animals (n=35) and no defects in the postembryonic migrations of the distal tip cells (DTCs) or male linker cell were seen in dpy-19(mu78) animals (n=50)."Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Variation_effectNullPaper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
EQ_annotationsAnatomy_termWBbt:0006830PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
GO_termGO:0016477PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
WBPhenotype:0000471Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Remark"No defects in the embryonic migrations of HSN, CAN, M and ALM were seen in dpy-19(n1347n1348) animals (n=35) and no defects in the postembryonic migrations of the distal tip cells (DTCs) or male linker cell were seen in dpy-19(mu78) animals (n=50)."Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Variation_effectNullPaper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
EQ_annotationsAnatomy_termWBbt:0005406PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
GO_termGO:0016477PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
WBPhenotype:0000594Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Remark"No defects in the embryonic migrations of HSN, CAN, M and ALM were seen in dpy-19(n1347n1348) animals (n=35) and no defects in the postembryonic migrations of the distal tip cells (DTCs) or male linker cell were seen in dpy-19(mu78) animals (n=50)."Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Variation_effectNullPaper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
EQ_annotationsAnatomy_termWBbt:0004489PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
GO_termGO:0016477PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
WBPhenotype:0001511Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Remark"No defects in the embryonic migrations of HSN, CAN, M and ALM were seen in dpy-19(n1347n1348) animals (n=35) and no defects in the postembryonic migrations of the distal tip cells (DTCs) or male linker cell were seen in dpy-19(mu78) animals (n=50)."Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
Variation_effectNullPaper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
EQ_annotationsAnatomy_termWBbt:0005062PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
GO_termGO:0016477PATO:0000460Paper_evidenceWBPaper00004437
Curator_confirmedWBPerson2987
ReferenceWBPaper00004437
RemarkCis double mutant
MethodAllele