WormBase Tree Display for Interaction: WBInteraction000536139
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| WBInteraction000536139 | Interaction_type | Genetic | Mutual_enhancement | ||
|---|---|---|---|---|---|
| GI_module_one | Cis_phenotypic | ||||
| GI_module_two | Enhancing | ||||
| Interactor | Interactor_overlapping_gene | WBGene00000440 | Interactor_type | Non_directional | |
| WBGene00000435 | Interactor_type | Non_directional | |||
| Variation_interactor | WBVar00054467 | Interactor_type | Non_directional | ||
| WBVar00091303 | Interactor_type | Non_directional | |||
| Interaction_summary | "We then scored the extent of ALA axon migration and found that the ceh-10(rf) animals showed a ceh-17-like truncation of the ALA axons, albeit less severe (Fig 6B,C). While the ceh-17(lf) axon migration phenotype is similar when scored in either L1 or L2 stage animals, we found that a greater fraction of ceh-10(rf) ALA axons reach the tail by the L2 stage than at L1. This was surprising, as wild-type ALA axons complete their migration before hatching (Pujol et_al, 2000). To determine if the postembryonic ALA axon migration seen in ceh-10(rf) mutant animals might be dependent on CEH-17 activity, we examined ALA axon migration in ceh-17(np1);ceh-10(ct78) animals. We found the extent of axon migration to be similar between the L1 and the L2 stages, and more severe at each stage than in either single mutant (Fig 6C)." | ||||
| Throughput | Low_throughput | ||||
| Interaction_phenotype | WBPhenotype:0001224 | ||||
| Paper | WBPaper00036308 | ||||
| Remark | Figure 6C |
